The rate of alcohol oxidation can be limited by the transfer of reducing equivalents into mitochondria or by the actual capacity of the respiratory chain to oxidize these reducing equivalents. Shuttle capacity may become limiting under fasting metabolic states as the levels of shuttle components decrease. This may contribute to the lower rates of alcohol oxidation (in addition to lower ADH content) in the fasting metabolic state. Agents or conditions which enhance reoxidation of NADH by the respiratory chain can increase the rate of alcohol metabolism e.g. uncoupling agents can accelerate ethanol oxidation in the fed metabolic state (38,39). Although light-to-moderate alcohol consumption may not pose a risk to patients with CKD, the patients’ condition needs to be considered. Can kidneys recover from alcohol damage? Some individuals put social pressure upon others to drink to justify their own intake and you do not need to give in to this pressure. Ethyl alcohol and water are the main ingredients of alcohol beverages, but we cannot ignore other bioactivators in liquors, such as polyphenols. Not all forms of kidney disease are does drinking beer hurt your kidneys preventable, but adopting a healthful lifestyle may reduce the risk of complications, even in people […]